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Rabbit Superoxide Dismutases (SOD) ELISA Kit-Sandwich

Cat. No.EK3F251

Product TypeAnimal Immunoassay Kits

Size

Product Overview

BioVenic Rabbit Superoxide Dismutases (SOD) ELISA Kit-Sandwich is designed for the quantitative determination of Rabbit SOD in serum, plasma, tissue homogenate, cell culture supernatant, cell lysate, and other biological fluids using a Sandwich ELISA method. For research use only.

Specifications

Assay Type ELISA-Sandwich
Specificity The assay kit is specific for Rabbit SOD
Target Species Rabbit
Species Reactivity Rabbit
Detection Range 0.16-10 ng/mL
Reproducibility Intra-Assay: CV < 10%; Inter-Assay: CV < 12%
Assay Time Around 90 min
Sample Requirement Serum, plasma, tissue homogenate, cell culture supernatant, cell lysate, and other biological fluids.

Target Information

Superoxide dismutase [Cu-Zn] neutralizes radicals typically generated within cells that are toxic to biological systems. It is encoded by the SOD1 gene. This protein is a cytosolic, dimeric, carbohydrate-free molecule found ubiquitously, and is part of a family of isoenzymes involved in scavenging superoxide anions. In a rabbit cardiac ischemia-reperfusion model, SOD use significantly improves myocardial function and structure and reduces cardiomyocyte apoptosis and necrosis. Exogenous SOD protects against superoxide anion damage and delays atherosclerosis progression in a rabbit model of atherosclerosis.

Target/Biomarker Rabbit SOD
Target Synonym Superoxide Dismutases
Gene ID 100009313
UniProt ID P09212

Shipping and Storage

This product is shipped with gel ice packs. It is recommended to store at 2-8 °C (Up to 6 months).

Documents

COA

To request a Certificate of Analysis, please enter the Lot No. in the search box. Note: Certificate of Analysis not available for kits.

The product is for research use only.
Not for commercial, prophylactic, diagnostic, or therapeutic applications.

References

  1. Mondola, Paolo et al. "The Cu, Zn Superoxide Dismutase: Not Only a Dismutase Enzyme." Frontiers in physiology vol. 7 594. 29 Nov. 2016.
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