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Mouse Anti-Porcine H2.0-Like Homeobox Protein (HLX1) Monoclonal Antibody

Cat. No.VD12N396

Product TypeAnimal-targeted Antibodies

Size 100 μL

Product Overview

BioVenic mouse monoclonal antibody is specific for porcine H2.0-like homeobox protein. It is purified by affinity chromatography. It can be applied to WB assay of porcine H2.0-like homeobox protein.

Specifications

Application WB
Clonality Monoclonal
Classification Primary Antibody
Clone M5N10
Host Mouse
Target Species Porcine
Species Reactivity Porcine
Specificity Porcine H2.0-Like Homeobox Protein
Immunogen Recombinant protein of H2.0-like homeobox protein
Purification Purified by Affinity Chromatography
Concentration 1 mg/mL
Conjugation Unconjugated
Preservative and Stabilizer 0.02% Sodium Azide
Buffer 0.1M Tris-Glycine, pH 7.4, with 150 mM NaCl and 50% glycerol
Physical State Liquid

Target Information

Porcine H2.0-Like Homeobox Protein (HLX) is a transcription factor involved in the regulation of hematopoiesis and embryonic development. It plays a crucial role in the differentiation of T-helper cells and the maintenance of hematopoietic stem cells. HLX is also implicated in angiogenesis and adipogenesis, regulating the switch from white to brown fat through interactions with other transcription factors. Additionally, HLX has been identified as a potential therapeutic target in acute myeloid leukemia (AML) due to its role in promoting abnormal proliferation and differentiation block in myeloid progenitors.

Target Porcine H2.0-Like Homeobox Protein
Target Synonym HLX1
Gene ID 100517234
UniProt ID A0A5G2R5H9

Shipping and Storage

This product is shipped with wet ice packs. Store at -20°C on receipt (up to 12 months). Avoid repeated freezing and thawing as this may denature the antibody.

Documents

COA

To request a Certificate of Analysis, please enter the Lot No. in the search box. Note: Certificate of Analysis not available for kits.

The product is for research use only.
Not for commercial, prophylactic, diagnostic, or therapeutic applications.

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