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Mouse Anti-Porcine Macrophage Colony-Stimulating Factor 1 Receptor (CSF1R) Monoclonal Antibody-[APC]

Cat. No.VD11N989

Product TypeAnimal-targeted Antibodies

Product Overview

BioVenic mouse monoclonal antibody is specific for porcine macrophage colony-stimulating factor 1 receptor. It can be applied to FCM assay of porcine macrophage colony-stimulating factor 1 receptor.

Specifications

Application FCM
Clonality Monoclonal
Classification Primary Antibody
Clone J22N32
Host Mouse
Target Species Porcine
Species Reactivity Porcine
Specificity Porcine Macrophage Colony-Stimulating Factor 1 Receptor
Isotype IgG2
Immunogen Recombinant protein of macrophage colony-stimulating factor 1 receptor
Conjugation APC
Preservative and Stabilizer Sodium Azide
Buffer Phosphate Buffered Saline, 1% BSA
Physical State Lyophilized

Target Information

Porcine macrophage colony-stimulating factor 1 receptor (CSF1R) is a receptor tyrosine kinase essential for the development, survival, and function of macrophages and monocytes. It binds to its ligands, such as CSF1 and IL-34, initiating signaling cascades that promote cell proliferation, differentiation, and survival. In pigs, CSF1R is expressed on macrophages and is involved in regulating the mononuclear phagocyte system, which plays a crucial role in immune responses and tissue homeostasis.

Target Porcine Macrophage Colony-Stimulating Factor 1 Receptor
Target Synonym CSF1R
Gene ID 100517086
UniProt ID A0A5G2QIU0

Shipping and Storage

This product is shipped with wet ice packs. Store at -20°C to -80°C on receipt (up to 12 months). Avoid repeated freezing and thawing as this may denature the antibody.

Documents

COA

To request a Certificate of Analysis, please enter the Lot No. in the search box. Note: Certificate of Analysis not available for kits.

The product is for research use only.
Not for commercial, prophylactic, diagnostic, or therapeutic applications.

References

  1. Álvarez, Belén, et al. "Porcine macrophage markers and populations: an update." Cells 12.16 (2023): 2103.
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