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Mouse Anti-Equine MLANA Monoclonal Antibody

Cat. No.VD8Y5

Product TypeAnimal-targeted Antibodies

Size

Product Overview

BioVenic mouse anti-equine MLANA monoclonal antibody is designed to recognize equine melan-A protein. The antibody can be used in the application of flow cytometry, immunofluorescence, and immunohistochemistry. The antibody can be used to identify melanocytic lesions and to better understand the underlying mechanisms of melanoma development and progression in horses.

Specifications

Application FC; IF; IHC
Clonality Monoclonal Antibody
Classification Primary Antibody
Clone A7Y2
Host Mouse
Target Species Equine
Species Reactivity Equine; Canine
Specificity Equine Melan-A
Isotype IgG2b
Purification Protein A/G Chromatography Purified
Purity ≥ 90% (SDS-PAGE)
Conjugation Unconjugated
Preservative and Stabilizer 0.05% BSA, 0.05% Sodium azide (NaN3)
Buffer Phosphate buffered saline
Physical State Liquid

Target Information

Melan-A is a protein associated with melanocytes, the pigment-producing cells responsible for melanin production. Melan-A is a useful marker for studying melanocytes and melanoma in horses and can aid in the diagnosis and management of equine melanocytic lesions.

Target Equine Melan-A
Target Synonym MLANA; Melan-A
Gene ID 100051824
UniProt ID F6Y2L7

Shipping and Storage

This product is shipped with gel ice packs. It is recommended to aliquot and store at -20°C on receipt. (up to 12 months). Avoid repeated freezing and thawing as this may denature the antibody. Storage in frost-free freezers is not recommended.

Documents

COA

To request a Certificate of Analysis, please enter the Lot No. in the search box. Note: Certificate of Analysis not available for kits.

The product is for research use only.
Not for commercial, prophylactic, diagnostic, or therapeutic applications.

References

  1. Goh, Myeong-Jin, et al. "Depigmentation effect of kadsuralignan f on melan-a murine melanocytes and human skin equivalents." International Journal of Molecular Sciences 14.1 (2013): 1655-1666.
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